In MS, slowly expanding lesions were linked to more myelin loss in brain regions of damage and in normal-appearing brain ...
"The primary motivation behind our recent paper was a very speculative idea that myelin may have evolved as a highly ...
How does our nervous system operate so quickly and efficiently? The answer lies in a membranous structure called myelin. Aa Aa Aa All our activities — eating, walking, talking — are controlled ...
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Experimental drug speeds up myelin repair, restoring vision in miceThe drug, LL-341070, enhances the brain's ability to repair damaged myelin— the protective sheath around nerve fibers. Damage to myelin is a hallmark of diseases like MS, as well as a natural ...
You disconnect your cell phone from its charger as you get ready for a busy day. But even though it has been connected all night, the battery is only partially charged. You look at ...
A discovery led by OHSU was made possible by years of study conducted by University of Portland undergraduates. Scientists ...
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News Medical on MSNNew MS research harnessing the brain's healing powerA new multiple sclerosis (MS) research project designed to help the brain repair itself by activating its own healing ...
MS is a neurological disease. It causes the immune system to attack and destroy myelin, a protective fatty tissue surrounding nerve cells. This process, called demyelination, affects the function ...
We have recently discovered that mechanical forces generated by the extracellular matrix and other cells are also essential for correct myelination, and we are actively pursuing the molecular ...
Ocrevus treatment was linked to increases in myelin and reductions in iron in MS patients with paramagnetic rim lesions, per ...
Tocilizumab and rituximab reduce relapse rates and disability progression in NMOSD, with tocilizumab provides extra benefits in MOGAD.
CAMBRIDGE, Mass., February 11, 2025--(BUSINESS WIRE)--Cognito Therapeutics, a neurotechnology company advancing disease-modifying therapies to treat CNS diseases, today announced the publication ...
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